Patients with abnormal liver feature test results go to boosted threat for hepatotoxicity and also bone tissue marrow reductions (view CAUTIONS). Therapy needs to be stopped if liver enzymes are significantly increased or if scientifically significant reductions in red blood cell counts occur.

Although single doses of albendazole have been shown not to inhibit theophylline metabolism (view Drug Interactions), albendazole does cause cytochrome P450 1A in human hepatoma cells. Therefore, it is recommended that plasma focus of theophylline be kept an eye on throughout and after therapy with Albenza.

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Steady-state trough concentrations of albendazole sulfoxide were regarding 56 % higher when 8 milligrams dexamethasone was coadministered with each dose of albendazole (15 mg/kg/day) in 8 neurocysticercosis clients.

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In the fed state, praziquantel (40 mg/kg) boosted mean maximum plasma attention and area under the curve of albendazole sulfoxide by regarding 50 % in healthy and balanced subjects (n = 10) contrasted with a separate group of topics (n = 6) provided albendazole alone. Mean Tmax and mean plasma removal half-life of albendazole sulfoxide were unmodified. The pharmacokinetics of praziquantel were unmodified complying with coadministration with albendazole (400 milligrams).

Albendazole sulfoxide attentions in bile and also cystic liquid were increased (concerning 2-fold) in hydatid cyst individuals treated with cimetidine (10 mg/kg/day) (n = 7) compared with albendazole (20 mg/kg/day) alone (n = 12). Albendazole sulfoxide plasma concentrations were the same 4 hrs after dosing.

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